HCT-116人结肠癌细胞（附
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人结肠癌细胞HCT-116
diklerent tumor microenvironments (TME). This means that the eklicacy of immunotherapy in actual applications will, therefore, have diklerent results. Existing tumor immune subtype studies have mostly focused on immune cells, stromal cells, genes and molecules without considering the presence of microbes. Some studies have shown that
accelerated approach to introducing these therapies into the clinic to treat patients with esophageal cancer.As the first compartment of the protein secretory pathway, the endoplasmic reticulum (ER) acts as a protein synthesis factory, maintaining proteostasis and ER homeostasis. However, a variety of intrinsic and extrinsic perturbations, such as cancer, can disrupt the
| 种属 | 人 |
| 别称 | HCT-116; HCT.116; HCT_116; HCT116; CoCL2 |
| 组织来源 | 结肠 |
| 疾病 | 结直肠癌 |
| 传代比例/细胞消化 | 1：2-1:3传代 ，消化1-2分钟 ， |
| 培养基配置 | McCoy’s 5A 培养基；10%胎牛血清；1%双抗 |
| 简介 | HCT116是1979年M.Brattain等从患结肠癌的男性病人中分离的三株恶性细胞之一。在半固体琼脂糖培养基中形成克 隆。HCT116在无胸腺的裸鼠有致瘤性 ，形成上皮样的肿瘤 |
| 形态 | 上皮细胞样 |
| 生长特征 | 贴壁生长 |
| 倍增时间 | ~48h |
| 基因表达 | carcinoembryonic antigen (CEA) 1 ng per 1×10^6 cells per 10 days |
| 致瘤性 | Yes, in nude mice |
| STR | Amelogenin：X ，Y；CSF1PO：7 ，10；D13S317：10 ，12；D16S539：11 ，13；D18S51：16 ，17； D19S433：12 ，13；D21S11：29 ，30；D2S1338：16；D3S1358：12 ，18 ，19；D5S818：10 ，11； D7S820：11 ，12；D8S1179：12 ，14；FGA：18 ，23；TH01：8 ，9；TPOX：8；vWA：17 ，22； |
| 保藏机构 | ATCC; CCL-247 |
diklerent tumor microenvironments (TME). This means that the eklicacy of immunotherapy in actual applications will, therefore, have diklerent results. Existing tumor immune subtype studies have mostly focused on immune cells, stromal cells, genes and molecules without considering the presence of microbes. Some studies have shown that
accelerated approach to introducing these therapies into the clinic to treat patients with esophageal cancer.As the first compartment of the protein secretory pathway, the endoplasmic reticulum (ER) acts as a protein synthesis factory, maintaining proteostasis and ER homeostasis. However, a variety of intrinsic and extrinsic perturbations, such as cancer, can disrupt the
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